HYPERACTIVITY BLEND 30ML

DIRECTIONS FOR TOPICAL USE

Apply a small amount to appropriate intact skin and massage gently.

Suitable areas include:

Underneath Feet

Upper back , neck and chest

Shoulders and inhalation

For evening use, apply before the child’s normal bedtime routine.

For daytime support, use conservatively before periods where calm attention and behavioural regulation are needed.

HYPERACTIVITY BLEND 30ML

Avoid the face, eyes, mouth, nose, mucous membranes and broken skin.

Wash adult hands after applying the product to a child.

Patch test before first use.

HYPERACTIVITY BLEND 30ML

Lavender

Mandarin/Tangerine

Roman Chamomile

Frankincense

Sweet Orange

Vetiver

Sandalwood

Bergamot

Cedarwood

Valerian and Olive Oil.

Advanced Botanical Neuroregulation

Hyperactivity Support has been designed around neurochemical regulation rather than simple sedation.

Linalool and Linalyl Acetate from Lavender influence excitatory glutamatergic/NMDA signalling, serotonin transport and neuronal ion-channel activity.

Valerian-derived Valerenic Acid provides a complementary inhibitory mechanism through allosteric modulation of GABA-A receptors.

Roman Chamomile adds ester-rich neuromuscular regulation.

Vetiver, Cedarwood and Sandalwood provide a dense sesquiterpene neuroregulatory fraction.

Bergamot, Mandarin and Sweet Orange contribute limonene-rich chemistry supporting autonomic and stress-response regulation.

Frankincense completes the formula with a terpene-rich neurological and inflammatory-support component.

HYPERACTIVITY BLEND 30ML

Hyperactivity Support is a specialised neuroregulatory botanical formulation developed to support children and adults experiencing patterns of excessive physical restlessness, heightened arousal, difficulty settling, sensory overstimulation, emotional reactivity and difficulty transitioning into rest and sleep.

The formulation combines:

Lavender • Mandarin/Tangerine • Roman Chamomile • Frankincense • Sweet Orange • Vetiver • Sandalwood • Bergamot • Cedarwood • Valerian • Olive Oil

Rather than functioning through one pathway, the formulation combines volatile phytochemicals with activity relevant to several areas of central nervous-system physiology:

GABAergic inhibitory signalling

Glutamatergic/NMDA excitatory signalling

Serotonergic signalling

Neuronal membrane excitability

Autonomic nervous-system regulation

Stress-response pathways

Sleep and arousal regulation

The pharmacological objective is not simply sedation.

It is to support the neurological balance between excitation and inhibition—helping an overstimulated nervous system move toward a more regulated physiological state.

UNDERSTANDING HYPERACTIVITY

Hyperactivity is not simply “too much energy.”

In conditions such as ADHD, the neurobiology is considerably more complex and can involve altered regulation within networks responsible for:

Attention
Impulse control
Executive function
Working memory
Motivation
Behavioural inhibition
Emotional regulation

Catecholamine signalling—particularly dopamine and noradrenaline within prefrontal cortical networks—is important in ADHD.

But behavioural hyperarousal is also influenced by the broader balance between excitatory and inhibitory neurotransmission.

Two important neurotransmitter systems are:

GLUTAMATE

The brain’s principal excitatory neurotransmitter.

GABA

The principal inhibitory neurotransmitter.

An effective calming formulation should therefore not be described simply as “making a child sleepy.”

The pharmacological concept is reducing excessive neuronal arousal while supporting a calmer state in which attention, emotional control and sleep can function more effectively.

LAVENDER

Linalool & Linalyl Acetate — CNS Excitability and Glutamate Regulation

Lavender forms the principal component of Hyperactivity Support.

Its major pharmacologically active volatile constituents are:

LINALOOL

LINALYL ACETATE

These compounds have been extensively investigated in central nervous-system pharmacology.

Lavender and linalool interact with several neuronal targets relevant to hyperarousal.

Experimental research demonstrates activity involving:

NMDA/glutamate signalling

Serotonin transporter — SERT

Voltage-dependent calcium channels

Voltage-dependent sodium channels

Neuronal excitability

Lavender essential oil and its principal constituents have demonstrated affinity for the NMDA glutamate receptor complex.

Linalool has also demonstrated inhibition of glutamate-related signalling.

Because glutamate is the brain’s principal excitatory neurotransmitter, modulation of excessive glutamatergic activity provides a plausible mechanism through which Lavender can reduce neuronal hyperexcitability. (PubMed)

Linalool can additionally influence voltage-dependent sodium channels, while Lavender oil has demonstrated effects on calcium influx through voltage-dependent calcium channels.

These channels participate directly in neuronal firing and neurotransmitter release.

Lavender and linalool have also demonstrated interaction with the serotonin transporter, SERT, providing an additional serotonergic mechanism relevant to emotional regulation and anxiety. (PubMed)

PRINCIPAL ACTIVE COMPONENTS

Linalool • Linalyl Acetate

PRIMARY PHARMACOLOGICAL RELEVANCE

Glutamatergic/NMDA modulation • Neuronal excitability regulation • SERT interaction • Calcium-channel modulation • CNS calming

VALERIAN

Valerenic Acid & GABA-A Receptor Modulation

Valerian provides one of the most specific inhibitory-neurotransmitter mechanisms represented in the formula.

The medicinal plant Valeriana officinalis contains compounds including:

VALERENIC ACID

VALERENOL

ACETOXYVALERENIC ACID

Valerenic acid has been demonstrated to act as an allosteric modulator of GABA-A receptors.

GABA-A receptors are chloride channels.

When GABA activates these receptors, chloride conductance contributes to neuronal hyperpolarisation, making neurons less likely to fire excessively.

Experimental research demonstrated that valerenic acid potentiated GABA-A receptor currents, particularly receptors containing β2 or β3 subunits. (PubMed)

Further research demonstrated that valerenic acid and valerenol act through GABA-A receptors containing the β3 subunit, providing a defined molecular mechanism for their anxiolytic activity in experimental models. (PubMed)

The pathway can therefore be represented as:

Valerenic acid → GABA-A receptor modulation → increased inhibitory signalling → reduced neuronal excitability

PRINCIPAL ACTIVE COMPONENTS

Valerenic Acid • Valerenol and related sesquiterpenic constituents

PRIMARY PHARMACOLOGICAL RELEVANCE

GABA-A modulation • Inhibitory neurotransmission • Hyperarousal reduction • Sleep physiology • Anxiolytic mechanisms

Valerian therefore provides an important inhibitory counterbalance within the formulation.

ROMAN CHAMOMILE

Ester-Rich Neuro-Muscular Regulation

Roman Chamomile — Chamaemelum nobile — contains a distinctive ester-rich volatile fraction.

Its chemistry commonly includes various angelate esters, with exact composition depending upon source.

These compounds contribute to Roman Chamomile’s pharmacological profile involving:

Smooth-muscle relaxation
Antispasmodic activity
Inflammatory regulation
Central calming effects

Roman Chamomile complements Lavender and Valerian by supporting reduction of both physical tension and neurological hyperarousal.

PRINCIPAL ACTIVE FRACTION

Angelate-rich volatile esters

PRIMARY PHARMACOLOGICAL RELEVANCE

Neuromuscular relaxation • Antispasmodic activity • Nervous-system calming • Physical restlessness support

VETIVER

Sesquiterpene-Rich Nervous-System Support

Vetiver — Chrysopogon zizanioides — is chemically very different from citrus or Lavender oils.

Its essential oil contains a dense sesquiterpene and sesquiterpene-alcohol fraction that can include compounds such as:

Khusimol
Vetiverol-related compounds
Vetivones
Khusimene and related sesquiterpenes

Its heavier sesquiterpene chemistry gives Vetiver a distinct pharmacological profile.

Experimental research has investigated Vetiver essential oil for effects on central nervous-system activity, alertness, behaviour and neurological function.

Within Hyperactivity Support, Vetiver is positioned as part of the attention-state and nervous-system-regulation complex.

PRINCIPAL ACTIVE FRACTION

Khusimol • Vetivones • Sesquiterpene alcohols and related sesquiterpenes

PRIMARY PHARMACOLOGICAL RELEVANCE

CNS regulation • Behavioural-state support • Neurophysiological stabilisation

CEDARWOOD

Sesquiterpene-Rich Neuroregulatory Component

Cedarwood provides another sesquiterpene-dominant component.

Its chemistry depends greatly on which botanical species is used.

For example, Cedrus atlantica commonly contains compounds from the himachalene family, while other materials marketed as Cedarwood can have very different chemistry.

Potential major constituents include:

β-Himachalene
α-Himachalene
γ-Himachalene
Atlantones

depending upon species and origin.

These sesquiterpenes give Cedarwood a fundamentally different chemical profile from the monoterpene-rich citrus oils.

Within Hyperactivity Support, Cedarwood provides a slower, heavier volatile fraction intended to complement Lavender, Vetiver, Valerian and Sandalwood within the formula’s CNS-regulatory architecture.

PRINCIPAL ACTIVE FRACTION

Himachalenes and related sesquiterpenes — species dependent

PRIMARY PHARMACOLOGICAL RELEVANCE

Neuroregulatory support • Stress-response modulation • Behavioural-state support

BERGAMOT

Limonene, Linalool & Linalyl Acetate

Bergamot contains a pharmacologically useful combination of:

LIMONENE

LINALYL ACETATE

LINALOOL

with other monoterpenes depending upon extraction.

This gives Bergamot both citrus-terpene and Lavender-like neuroactive chemistry.

Human and experimental research has investigated Bergamot in relation to stress physiology, anxiety, autonomic activity and mood.

Its linalool/linalyl acetate fraction complements Lavender’s effects on neuronal signalling, while limonene provides additional neurobiological and inflammatory activity.

PRINCIPAL ACTIVE COMPONENTS

Limonene • Linalyl Acetate • Linalool

PRIMARY PHARMACOLOGICAL RELEVANCE

Stress-response regulation • Autonomic modulation • CNS support • Emotional regulation

MANDARIN / TANGERINE

Limonene-Rich Regulation

Mandarin/Tangerine essential oil is generally dominated by:

D-LIMONENE

with smaller quantities of compounds such as:

γ-Terpinene
α-Pinene
β-Pinene
Myrcene

depending upon species and source.

Limonene has been investigated experimentally for neuroactive, antioxidant and inflammatory effects.

Within this formula Mandarin primarily supports the stress-arousal and emotional-regulation component, complementing the stronger CNS-active Lavender, Valerian and Vetiver fractions.

PRINCIPAL ACTIVE COMPONENT

D-Limonene

PRIMARY PHARMACOLOGICAL RELEVANCE

Stress-response support • Neurophysiological modulation • Antioxidant support

SWEET ORANGE

D-Limonene & Autonomic Stress Response

Sweet Orange also provides a predominantly:

D-LIMONENE

volatile fraction.

Citrus-oil research has investigated effects on anxiety, stress responses and autonomic nervous-system activity.

Sweet Orange therefore strengthens the formula’s stress-arousal regulation rather than serving as a direct sedative.

This distinction is important.

A child who is restless because of physiological hyperarousal does not necessarily need to be heavily sedated.

The objective is to support movement from an excessively activated state toward a more regulated state.

SANDALWOOD

α-Santalol & β-Santalol

Sandalwood provides two particularly important sesquiterpene alcohols:

α-SANTALOL

β-SANTALOL

These compounds have been investigated for effects involving central nervous-system activity, inflammatory signalling and cellular physiology.

Sandalwood therefore adds another sesquiterpene-rich component to the Cedarwood–Vetiver–Sandalwood axis of the formula.

PRINCIPAL ACTIVE COMPONENTS

α-Santalol • β-Santalol

PRIMARY PHARMACOLOGICAL RELEVANCE

Neurophysiological regulation • CNS calming • Inflammatory modulation

FRANKINCENSE

α-Pinene & Terpene-Mediated Neurological Support

Frankincense essential oil contains a species-dependent mixture of volatile terpenes.

These can include:

α-Pinene
α-Thujene
Limonene
Sabinene
β-Caryophyllene and other terpenes

depending upon Boswellia species and origin.

α-Pinene has been investigated experimentally for neuroprotective, inflammatory and neurotransmitter-related effects.

Frankincense therefore contributes to the formula’s neurological and inflammatory-support architecture.

PRINCIPAL ACTIVE FRACTION

α-Pinene and related Boswellia volatile terpenes

PRIMARY PHARMACOLOGICAL RELEVANCE

Neuroprotective support • Inflammatory regulation • CNS support

THE NEUROPHARMACOLOGICAL STRATEGY

Hyperactivity Support can be understood through four interconnected systems.

1. EXCITATORY NEUROTRANSMISSION

LAVENDER — LINALOOL & LINALYL ACETATE

supports regulation of glutamatergic/NMDA signalling and neuronal ion-channel activity. (PubMed)

This addresses one side of neuronal excitability:

EXCESSIVE EXCITATORY SIGNALLING

2. INHIBITORY NEUROTRANSMISSION

VALERIAN — VALERENIC ACID

acts as an allosteric modulator of specific GABA-A receptors, enhancing inhibitory neuronal signalling in experimental systems. (PubMed)

This addresses the opposite side:

INSUFFICIENT INHIBITORY CONTROL / EXCESSIVE AROUSAL

Together, Lavender and Valerian therefore provide a particularly logical neurochemical pairing:

GLUTAMATE ↓ / MODULATION

GABA ↑ / POTENTIATION

=

REDUCED NEURONAL HYPEREXCITABILITY

This is a pharmacological concept rather than simply an aromatherapy description.

3. STRESS & AUTONOMIC REGULATION

Lavender
Bergamot
Mandarin
Sweet Orange
Roman Chamomile

provide overlapping phytochemistry relevant to stress, autonomic arousal and emotional regulation.

This is particularly useful where hyperactive behaviour becomes worse during:

Anxiety
Sensory overload
Emotional stress
Transitions
Fatigue
Overstimulation

4. CNS & BEHAVIOURAL-STATE SUPPORT

Vetiver
Cedarwood
Sandalwood
Frankincense

provide a sesquiterpene-rich component complementing the more volatile linalool- and limonene-rich oils.

The formula therefore combines:

MONOTERPENE CNS ACTIVITY

with

SESQUITERPENE NEUROREGULATORY SUPPORT

rather than depending upon one chemical family.

HOW HYPERACTIVITY SUPPORT MAY SUPPORT THE CHILD

The intended physiological objective is to support:

Reduced excessive arousal

Improved ability to settle

Reduced physical restlessness

Better transition from activity into quiet tasks

Emotional regulation

Reduced sensory overstimulation

Calmer bedtime transition

Sleep preparation

A more regulated state in which attention is easier to sustain

The distinction is important:

CALMING IS NOT THE SAME AS SEDATING.

The objective during the day is not to make the child sleepy.

It is to reduce unnecessary neurological and physiological “noise” so that the brain can function from a more regulated state.

DAYTIME SUPPORT

During the day, Hyperactivity Support can be incorporated before situations requiring greater regulation, such as:

School preparation
Homework
Quiet learning
Long journeys
Transitions between activities
Periods of overstimulation

The objective is a calmer but functional neurological state.

EVENING & SLEEP SUPPORT

The formulation also contains a strong sleep-transition component.

Lavender and Valerian are particularly relevant here.

Valerenic acid’s interaction with GABA-A receptors provides a defined molecular rationale for its role in reducing CNS arousal. (PubMed)

Lavender contributes complementary effects on glutamatergic, serotonergic and neuronal ion-channel systems. (DOI)

Together with Roman Chamomile, Vetiver, Cedarwood and Sandalwood, this provides a multi-pathway approach to helping the nervous system transition from:

HIGH AROUSAL → REGULATION → REST → SLEEP

DIRECTIONS FOR TOPICAL USE

Apply a small amount to appropriate intact skin and massage gently.

Suitable areas include:

underneath Feet • Upper back , neck and chest  • Shoulders and inhalation 

For evening use, apply before the child’s normal bedtime routine.

For daytime support, use conservatively before periods where calm attention and behavioural regulation are needed.

Avoid the face, eyes, mouth, nose, mucous membranes and broken skin.

Wash adult hands after applying the product to a child.

Patch test before first use.

PRIMARY PHARMACOLOGICAL TARGETS

GABA-A signalling
Glutamatergic/NMDA signalling
Serotonergic signalling
Voltage-dependent ion channels
Neuronal excitability
Autonomic stress regulation
Sleep-arousal physiology

KEY BIOACTIVE COMPOUNDS

Linalool / Linalyl Acetate — Lavender

Valerenic Acid / Valerenol-related chemistry — Valerian preparations

Angelate esters — Roman Chamomile

Khusimol / Vetivones / Sesquiterpenes — Vetiver

Himachalenes — Cedarwood, species dependent

α-Santalol / β-Santalol — Sandalwood

Limonene / Linalool / Linalyl Acetate — Bergamot

D-Limonene — Mandarin/Tangerine and Sweet Orange

α-Pinene and related terpenes — Frankincense

INGREDIENTS

Lavender, Mandarin/Tangerine, Roman Chamomile, Frankincense, Sweet Orange, Vetiver, Sandalwood, Bergamot, Cedarwood, Valerian and Olive Oil.

HYPERACTIVITY SUPPORT

Advanced Botanical Neuroregulation

Hyperactivity Support has been designed around neurochemical regulation rather than simple sedation.

Linalool and Linalyl Acetate from Lavender influence excitatory glutamatergic/NMDA signalling, serotonin transport and neuronal ion-channel activity.

Valerian-derived Valerenic Acid provides a complementary inhibitory mechanism through allosteric modulation of GABA-A receptors.

Roman Chamomile adds ester-rich neuromuscular regulation.

Vetiver, Cedarwood and Sandalwood provide a dense sesquiterpene neuroregulatory fraction.

Bergamot, Mandarin and Sweet Orange contribute limonene-rich chemistry supporting autonomic and stress-response regulation.

Frankincense completes the formula with a terpene-rich neurological and inflammatory-support component.

The resulting strategy is:

EXCESSIVE NEURONAL AROUSAL

↓

GLUTAMATE / NMDA MODULATION

Lavender — Linalool & Linalyl Acetate
+

INHIBITORY GABA-A SUPPORT

Valerian — Valerenic Acid
+

AUTONOMIC & STRESS REGULATION

Bergamot • Mandarin • Orange • Chamomile
+

CNS-REGULATORY SESQUITERPENES

Vetiver • Cedarwood • Sandalwood • Frankincense
↓

CALMER NEUROLOGICAL STATE

↓

BETTER SETTLING • EMOTIONAL REGULATION • REST • SLEEP SUPPORT

HYPERACTIVITY SUPPORT

Regulate excessive arousal.
Support inhibitory signalling.
Reduce neurological overstimulation.
Support emotional regulation.
Create the physiological conditions for calmer attention and restorative sleep.