Hyperthyroid Support (overactive Thyroid) 120 Capsules

Hyperthyroid Support (overactive Thyroid) 120 Capsules

R380.00 Incl. VAT

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In stock

Our natural herbal supplement is formulated to support thyroid health and alleviate symptoms associated with hyperthyroidism. This carefully crafted blend aims to normalize thyroid function, reduce inflammation, and address autoimmune aspects linked to thyroid issues.

With a focus on natural ingredients, our supplement offers a comprehensive approach to managing hyperthyroidism and promoting overall thyroid well-being.

Hyperthyroidism is a medical condition characterized by the overactivity of the thyroid gland, a butterfly-shaped gland, located in the front of the neck. The thyroid gland plays a crucial role in regulating the body’s metabolism by producing hormones, primarily thyroxine (T4) and triiodothyronine (T3). These hormones are essential for maintaining the balance of various bodily functions, including heart rate, energy production, and body temperature.

In hyperthyroidism, the thyroid gland produces an excessive amount of thyroid hormones, leading to an accelerated metabolic rate and an array of symptoms. The most common cause of hyperthyroidism is an autoimmune disorder called Graves’ disease, where the body’s immune system mistakenly attacks the thyroid gland, causing it to produce more hormones.

Symptoms of hyperthyroidism may include:

Weight loss: Despite increased appetite, individuals with hyperthyroidism often experience weight loss.

  • Increased heart rate: An elevated heart rate (tachycardia) and palpitations are common symptoms.
  • Nervousness and irritability: Hyperthyroidism can lead to increased anxiety, nervousness, and irritability.
  • Fatigue: Paradoxically, some people with hyperthyroidism may also experience fatigue and weakness.
  • Heat intolerance: Individuals may feel overly warm or have an increased sensitivity to heat.
  • Excessive sweating: Hyperthyroidism can cause an increase in perspiration.
  • Tremors: Fine tremors in the hands or fingers may occur.
  • Changes in menstrual patterns: Women with hyperthyroidism may experience irregular menstrual cycles.
  • Enlarged thyroid gland (goiter): In some patients, the thyroid gland may become visibly enlarged.
  • Eye problems: In Graves’ disease, there may be eye-related symptoms such as bulging eyes (exophthalmos) and eye irritation.

Diagnosis of hyperthyroidism involves blood tests to measure thyroid hormone levels (T3 and T4) and thyroid-stimulating hormone (TSH). Additional imaging studies, such as a thyroid scan or ultrasound, may be performed to assess the size and function of the thyroid gland.

Treatment options for hyperthyroidism may include medications to block the production of thyroid hormones, radioactive iodine therapy to reduce thyroid activity, or, in some cases, surgery to remove part, or the entire thyroid gland. Management is typically tailored to the underlying cause and the severity of symptoms. It is important for individuals experiencing symptoms of hyperthyroidism to seek medical attention for a proper diagnosis.

The Thyroid function in the brain:

The thyroid function is regulated by a complex feedback system involving the hypothalamus, the pituitary gland, and the thyroid gland. This system is often referred to as the hypothalamic-pituitary-thyroid (HPT) axis. Here’s an overview of the process:

  • Hypothalamus: The process begins in the hypothalamus, a region in the brain. The hypothalamus produces thyrotropin-releasing hormone (TRH) in response to signals from the body indicating the need for thyroid hormones. TRH then travels to the next component in the axis.
  • Pituitary Gland: TRH stimulates the pituitary gland, which is a small gland located at the base of the brain. In response to TRH, the pituitary gland releases thyroid-stimulating hormone (TSH) into the bloodstream.
  • Thyroid Gland: TSH acts on the thyroid gland located in the neck and is shaped like a butterfly. The thyroid gland produces and releases thyroid hormones, primarily thyroxine (T4) and triiodothyronine (T3), into the bloodstream. These hormones are crucial for the regulation of metabolism throughout the body.
  • Peripheral Tissues: T3 and T4 circulate in the bloodstream and reach various tissues and organs throughout the body. These hormones play a key role in regulating the metabolic rate, energy production, and the functioning of organs such as the heart and muscles.
  • Feedback Mechanism: As the levels of T3 and T4 in the bloodstream increase, they provide feedback to the hypothalamus and pituitary gland to inhibit the production and release of TRH and TSH. This feedback mechanism helps maintain a balance of thyroid hormones in the body.

In cases of hyperthyroidism, such as in Graves’ disease, an autoimmune disorder, the immune system produces antibodies that mimic TSH. These antibodies bind to the TSH receptors on the thyroid gland, leading to an overproduction of thyroid hormones. This disrupts the normal feedback mechanism, causing an excess of thyroid hormones in the bloodstream.

Conversely, hypothyroidism occurs when the thyroid gland does not produce thyroid hormones. This can result from various causes, such as autoimmune thyroiditis (Hashimoto’s disease), iodine deficiency, or certain medications. In hypothyroidism, the feedback mechanism prompts an increase in TRH and TSH production, in an attempt to stimulate the thyroid gland to produce more hormones.

Our natural herbal supplement is formulated to support thyroid health and alleviate symptoms associated with hyperthyroidism. This carefully crafted blend aims to normalize thyroid function, reduce inflammation, and address autoimmune aspects linked to thyroid issues.

With a focus on natural ingredients, our supplement offers a comprehensive approach to managing hyperthyroidism and promoting overall thyroid well-being.

Hyperthyroidism is a medical condition characterized by the overactivity of the thyroid gland, a butterfly-shaped gland, located in the front of the neck. The thyroid gland plays a crucial role in regulating the body’s metabolism by producing hormones, primarily thyroxine (T4) and triiodothyronine (T3). These hormones are essential for maintaining the balance of various bodily functions, including heart rate, energy production, and body temperature.

In hyperthyroidism, the thyroid gland produces an excessive amount of thyroid hormones, leading to an accelerated metabolic rate and an array of symptoms. The most common cause of hyperthyroidism is an autoimmune disorder called Graves’ disease, where the body’s immune system mistakenly attacks the thyroid gland, causing it to produce more hormones.

Symptoms of hyperthyroidism may include:

Weight loss: Despite increased appetite, individuals with hyperthyroidism often experience weight loss.

  • Increased heart rate: An elevated heart rate (tachycardia) and palpitations are common symptoms.
  • Nervousness and irritability: Hyperthyroidism can lead to increased anxiety, nervousness, and irritability.
  • Fatigue: Paradoxically, some people with hyperthyroidism may also experience fatigue and weakness.
  • Heat intolerance: Individuals may feel overly warm or have an increased sensitivity to heat.
  • Excessive sweating: Hyperthyroidism can cause an increase in perspiration.
  • Tremors: Fine tremors in the hands or fingers may occur.
  • Changes in menstrual patterns: Women with hyperthyroidism may experience irregular menstrual cycles.
  • Enlarged thyroid gland (goiter): In some patients, the thyroid gland may become visibly enlarged.
  • Eye problems: In Graves’ disease, there may be eye-related symptoms such as bulging eyes (exophthalmos) and eye irritation.

Diagnosis of hyperthyroidism involves blood tests to measure thyroid hormone levels (T3 and T4) and thyroid-stimulating hormone (TSH). Additional imaging studies, such as a thyroid scan or ultrasound, may be performed to assess the size and function of the thyroid gland.

Treatment options for hyperthyroidism may include medications to block the production of thyroid hormones, radioactive iodine therapy to reduce thyroid activity, or, in some cases, surgery to remove part, or the entire thyroid gland. Management is typically tailored to the underlying cause and the severity of symptoms. It is important for individuals experiencing symptoms of hyperthyroidism to seek medical attention for a proper diagnosis.

The Thyroid function in the brain:

The thyroid function is regulated by a complex feedback system involving the hypothalamus, the pituitary gland, and the thyroid gland. This system is often referred to as the hypothalamic-pituitary-thyroid (HPT) axis. Here’s an overview of the process:

  • Hypothalamus: The process begins in the hypothalamus, a region in the brain. The hypothalamus produces thyrotropin-releasing hormone (TRH) in response to signals from the body indicating the need for thyroid hormones. TRH then travels to the next component in the axis.
  • Pituitary Gland: TRH stimulates the pituitary gland, which is a small gland located at the base of the brain. In response to TRH, the pituitary gland releases thyroid-stimulating hormone (TSH) into the bloodstream.
  • Thyroid Gland: TSH acts on the thyroid gland located in the neck and is shaped like a butterfly. The thyroid gland produces and releases thyroid hormones, primarily thyroxine (T4) and triiodothyronine (T3), into the bloodstream. These hormones are crucial for the regulation of metabolism throughout the body.
  • Peripheral Tissues: T3 and T4 circulate in the bloodstream and reach various tissues and organs throughout the body. These hormones play a key role in regulating the metabolic rate, energy production, and the functioning of organs such as the heart and muscles.
  • Feedback Mechanism: As the levels of T3 and T4 in the bloodstream increase, they provide feedback to the hypothalamus and pituitary gland to inhibit the production and release of TRH and TSH. This feedback mechanism helps maintain a balance of thyroid hormones in the body.

In cases of hyperthyroidism, such as in Graves’ disease, an autoimmune disorder, the immune system produces antibodies that mimic TSH. These antibodies bind to the TSH receptors on the thyroid gland, leading to an overproduction of thyroid hormones. This disrupts the normal feedback mechanism, causing an excess of thyroid hormones in the bloodstream.

Conversely, hypothyroidism occurs when the thyroid gland does not produce thyroid hormones. This can result from various causes, such as autoimmune thyroiditis (Hashimoto’s disease), iodine deficiency, or certain medications. In hypothyroidism, the feedback mechanism prompts an increase in TRH and TSH production, in an attempt to stimulate the thyroid gland to produce more hormones.

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